So my wife has recently received her MRI report and we are not sure if we should be concerned or not. Our MD is out of the office for 8 weeks(!) so getting some insight is going to take a while. She has constant numbness on her right side and spends every day in pain, almost to a level where she cannot take it.
I am posting the results anonymously and if anyone out there can review and just give me their two cents, I would appreciate it. The mind can wreak havoc and at this point we are just looking for some piece of mind. Cheers.
MRI BRAIN AND CERVICAL SPINE
INDICATION: 41-year-old female, rule out multiple sclerosis. Constant numbness since September 2015 right hand and foot not explained by nerve conduction studies.
BRAIN: Multiplanar, multi sequence unenhanced images of the brain. No prior relevant MRI available.
The grey-white matter differentiation is maintained. No restricted effusion. No infra-axial or extra-axial mass or fluid collection. No abnormal parenchymal signal intensity. No thinning or abnormal signal intensity of the corpus callosum. Ventricles are symmetrical, no hydrocephalus. Intracranial flow voids are maintained. The imaged portion of orbits, sella/suprasellar region are unremarkable. The craniocervical junction is within normal limits.
CERVICAL SPINE: Multiplanar, multi sequence unenhanced and enhanced images of the brain. No prior relevant MRI available. Vertebral body height is maintained. Mild broad-based disc bulge at C5-C6 and C6-C7, no significant central canal or neural foraminal narrowing at these levels.
Centrally within the dorsal aspect of the cervical spinal cord non-enhancing T2 hyperintensity measuring up to 3 mm transverse by 2.5 mm cranial caudal extending from the level of the inferior aspect of C2 inferiorly to the the level of the superior aspect of T1 vertebral body, cannot be confirmed to be contiguous, in areas question to be non contiguous, no correlate evident on the Ti: sequence. No atrophic changes of the cervical spinal cord.
IMPRESSION:
No acute intracranial pathology evident. No findings to suggest intracranial demyelinating disease. Centrally within the dorsal cervical spinal cord nonenhancing T2 hyperintensity measuring up to 3 mm transverse by 2.5 mm cranial caudal extending from the level of inferior aspect of C2 inferiorly to the level of superior aspect of Ti vertebral body as described, of uncertain etiology. No prior imaging to determine stability/chronicity. Signal intensity changes appear more dorsal within the cervical spine than expected for syrinx. Findings are nonspecific, could be secondary to demyelinating disease, though not completely typical is there history to suggest subacute combined degeneration of the cord secondary to such etiology as B12 deficiency. Clinical correlation with short follow up MRI advised to confirm stability.